01 January 2015 by Kurt R Stenmark, Michael Yeager

The constitutively activated "imprinted" phenotype of fibroblasts is due, at least in part, to increased HDAC activity

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Stenmark {KR},Yeager M,Riddle S,El Kasmi {KC},Frid {MG},Li M,{McKinsey} T.Targeting the adventitial microenvironment in pulmonary hypertension: A potential approach to therapy that considers epigenetic change. [Internet]. 2012 ;2:3. Available from:http://www.pulmonarycirculation.org/text.asp?2012/2/1/3/94817

The constitutively activated "imprinted" phenotype of fibroblasts is due, at least in part, to increased HDAC activity.

 

Additional keywords: fibroblast,HDAC,inflammation,

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